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Abstract

Stanisław Jan Gąsiorowski (1897–1962) studied classical archaeology and art history at the Jagiellonian University during the years 1915 –1920, under direction of Piotr Bieńkowski and Julian Pagaczewski. During a one–year stay in Vienna, he attended lectures given by Joseph Strzygowski, Max Dvořák and Julius Schlosser. In 1922, he started his professional career as an assistant in the Chair of Classical Archaeology at the Jagiellonian University. In 1925, he obtained his doctorate and in 1928, he received his habilitation. In 1930 he was named profesor extraordinariusand in 1937 ordinarius. He remained in this position until 1953. In November of 1939, along with other professors of the Jagiellonian University, he was arrested by the Nazis, and imprisoned in the Sachsenhausen concentration camp. In 1940 he was released. In 1942 the Prince Czartoryski family entrusted him with the position of director of the Czartoryski Museum. In 1951, Prof. Gąsiorowski was dismissed, under the pretext that he was in the service of “aristocratic and bourgeois enemies of the Polish people”. Shortly thereafter the authorities also forced his removal from the University (1953). Deprived of the opportunity to give lectures and be in contact with students, he shifted his work to the Institute of Material Culture of the Polish Academy of Science, and remained there until his death. His research interests followed three general themes. The first of these was ancient art in the strict sense. One of Prof. Gąsiorowski’s great achievements was to write Poland’s first summary of the history of ancient art, from Egypt and the ancient Near East to Early Christian Art. The second area involved the theoretical foundations for the study of the material culture of Mediterranean countries, the relationships between art and material culture, and ergological classification. Finally, the third area was the publication of ancient and modern artworks from Polish collections as well as their history, and information on early Polish travelers to the Mediterranean countries.
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Abstract

Photovoltaic panels have a non-linear current-voltage characteristics to produce the maximum power at only one point called the maximum power point. In the case of the uniform illumination a single solar panel shows only one maximum power, which is also the global maximum power point. In the case an irregularly illuminated photovoltaic panel many local maxima on the power-voltage curve can be observed and only one of them is the global maximum. The proposed algorithm detects whether a solar panel is in the uniform insolation conditions. Then an appropriate strategy of tracking the maximum power point is taken using a decision algorithm. The proposed method is simulated in the environment created by the authors, which allows to stimulate photovoltaic panels in real conditions of lighting, temperature and shading.
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Abstract

Pancreatic ductal adenocarcinoma (PDAC) is characterized by very poor prognosis. It is caused by asymptomatic course of the disease at early stage. Symptomatic PDAC means usually advanced stage of the disease, making radical treatment impossible. Finding of biological PDAC marker could improve PDAC treatment through early diagnosis. In our study, we investigated two adipokines: omentin and chemerin concentration in PDAC, chronic pancreatitis (CP) and healthy individuals. We examined 27 PDAC patients, 10 CP patients and 36 controls. To determine concentration of adipokines we used ELISA immunoenzymatic assay. Level of both adipokines was increased when comparing control group to PDAC patients. Additionally, chemerin concentration in CP group was elevated comparing to control. To evaluate both adipokines as potential PDAC biomarkers we performed ROC analysis. Chemerin (AUC = 0.913) displayed better discriminant ability than omentin-1 (AUC = 0.73). Some authors believe that chemerin may promote tumour growth by stimulating angiogenesis and is supposed to be a factor recruiting mesenchymal stroma cells (MSC) in tumour regions. Omentin-1 can inhibit tumourigenesis by TP53 stimulation. On the other hand, according to some studies, omentin-1 may promote cancer proliferation via Akt signalling pathway. Results from our study showed signifi cantly elevated level of chemerin and omentin-1 in PDAC patients. Th erefore, w e believe that both investigated adipokines may provide promising and novel pharmacological insights for oncological diagnosis in the near future.
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